Recovery as an explanatory mechanism in the relation between acute stress reactions and chronic health impairment
Recovery as an explanatory mechanism in the relation between acute stress reactions and chronic health impairment
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DOI：
10.5271/sjweh.1053
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发表时间：
2006-12-01
影响因子：
6.3
通讯作者：
Sonnentag, Sabine
中科院分区：
文献类型：
--
作者：
Geurts, Sabine A. E.;Sonnentag, Sabine
这项贡献旨在揭示对压力工作特征的急性反应与员工长期健康之间关系的潜在机制。恢复是付出努力后心理生理放松的过程，被认为是这种关系中的重要环节。这种联系是根据努力、恢复和持续激活理论的假设来解释的。有人认为，当工作期间的恢复机会（内部恢复）不足时，工作后的恢复（外部恢复）就尤其必要。进一步认为，有两种情况可能会通过维持生理激活、长时间暴露于工作需求（长时间工作）和认知压力相关过程（例如沉思）来阻碍恢复过程。这些理论假设得到了先前实验室和现场研究的实证支持的证实。结论是，持续的生理激活和不完全恢复的慢性状况是慢性健康损害的重要途径。
This contribution aims at shedding light on the mechanisms that may underlie the relationship between acute reactions to stressful work characteristics and employee health in the long run. Recovery, a process of psychophysiological unwinding after effort expenditure, is considered a vital link in this relationship. This link is explained on the basis of assumptions from theories on effort, recovery, and sustained activation. It is argued that recovery after work (external recovery) is particularly necessitated when recovery opportunities during worktime (internal recovery) are insufficient. It is further argued that two conditions may impede the recovery process by sustaining physiological activation, prolonged exposure to work demands (working long hours) and cognitive stress-related processes (such as rumination). These theoretical assumptions are substantiated by empirical support from previous laboratory and field research. It is concluded that the chronic situation of sustained physiological activation and incomplete recovery is an important pathway to chronic health impairment.