Cathepsin H deficiency in mice induces excess Th1 cell activation and early-onset of EAE through impairment toll-like receptor 3 cascade.
Cathepsin H deficiency in mice induces excess Th1 cell activation and early-onset of EAE through impairment toll-like receptor 3 cascade.
复制标题
小鼠组织蛋白酶 H 缺乏会通过损害 Toll 样受体 3 级联来诱导 Th1 细胞过度激活和 EAE 早发。
作者：
Okada R;Zhang Z;Harada Y;Wu Z;Nakanishi H
