Lipid reactivity to stress: II. Biological and behavioral influences.
Lipid reactivity to stress: II. Biological and behavioral influences.
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脂质对应激的反应：II。
DOI：
10.1037//0278-6133.18.3.251
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发表时间：
1999
期刊：
影响因子：
--
通讯作者：
Flynn,M
中科院分区：
文献类型：
--
作者：
Stoney,CM;Bausserman,L;Niaura,R;Marcus,B;Flynn,M
本研究探讨了急性和慢性应激对血脂浓度的行为和生理影响。100名男性（n= 92）和女性（n= 8）进行了测试，在慢性应激和2急性应激。在慢性压力下，饮食，体力活动，锻炼和睡眠进行了检查。在急性应激，儿茶酚胺，皮质醇，血浆容量和心血管反应进行了检查。没有一种行为影响可以解释慢性压力下的脂质反应。致动脉粥样硬化脂质对急性应激的反应不仅反映了血浆的血液浓缩，而且与心血管、肾上腺素和皮质醇反应性中度相关。舒张压反应的急性应激源有较大的脂质反应的慢性应激比nonreactors。压力期间血脂升高不是人为因素，可能具有临床意义。（PsycINFO数据库记录（c）2016阿帕，保留所有权利）
This study examined behavioral and physiological influences on lipid concentrations during acute and chronic stressors. One hundred men (n= 92) and women (n= 8) were tested during a chronic stressor and during 2 acute stressors. During chronic stress, diet, physical activity, exercise, and sleep were examined. During the acute stressors, catecholamines, cortisol, plasma volume, and cardiovascular responses were examined. None of the behavioral influences could explain the lipid response to chronic stress. Responses of the atherogenic lipids to acute stressors were not solely reflecting hemoconcentration of the plasma but were moderately correlated with cardiovascular, epinephrine, and cortisol reactivity. Diastolic blood pressure reactors to the acute stressors had larger lipid responses to the chronic stressor than did nonreactors. Elevations in blood lipids during stress are not artifacts and may be clinically significant.(PsycINFO Database Record (c) 2016 APA, all rights reserved)