Interactions between propagating cracks and bioinspired self-healing vascules embedded in glass fibre reinforced composites
Interactions between propagating cracks and bioinspired self-healing vascules embedded in glass fibre reinforced composites
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DOI：
10.1016/j.compscitech.2011.01.027
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发表时间：
2011-04-12
影响因子：
9.1
通讯作者：
Trask, R. S.
中科院分区：
文献类型：
--
作者：
Norris, C. J.;Bond, I. P.;Trask, R. S.
本研究考虑将生物启发的脉管系统嵌入复合结构内，该复合结构能够将功能剂从外部储库递送到内部损伤区域。通过传播裂缝破坏血管，是激活这种自我修复系统的关键先决条件。两个隔离脉管制造技术证明，和他们的相互作用与传播模式I和II裂纹确定。血管的制造路线所采用的产生的层压体的形态，这反过来又决定了裂纹血管的相互作用发挥了重要作用。脉管的嵌入没有降低主层压板的模式I或II断裂韧性，脉管横向于裂纹扩展方向取向，导致通过裂纹止裂显著增加G（I）和G（II）。在模式II条件下，发现大的树脂袋会改变血管周围裂纹的方向，因此，建议避免这种配置用于自愈合应用。(C)2011爱思唯尔有限公司保留所有权利。
This study considers the embedment of a bioinspired vasculature within a composite structure that is capable of delivering functional agents from an external reservoir to regions of internal damage. Breach of the vascules, by propagating cracks, is a crucial pre-requisite for such a self-healing system to be activated. Two segregated vascule fabrication techniques are demonstrated, and their interactions with propagating Mode I and II cracks determined. The vascule fabrication route adopted played a significant role on the resulting laminate morphology which in-turn dictated the crack-vascule interactions. Embedment of the vascules did not lower the Mode I or II fracture toughness of the host laminate, with vascules orientated transverse to the crack propagation direction leading to significant increases in G(I) and G(II) through crack arrest. Large resin pockets were found to redirect the crack around the vascules under Mode II conditions, therefore, it is recommended to avoid this configuration for self-healing applications. (C) 2011 Elsevier Ltd. All rights reserved.