Radiosensitization by irinotecan is attributed to G2/M phase arrest, followed by enhanced apoptosis, probably through the ATM/Chk/Cdc25C/Cdc2 pathway in p53-mutant colorectal cancer cells
Radiosensitization by irinotecan is attributed to G2/M phase arrest, followed by enhanced apoptosis, probably through the ATM/Chk/Cdc25C/Cdc2 pathway in p53-mutant colorectal cancer cells
复制标题
伊立替康的放射增敏作用归因于 G2/M 期阻滞，随后细胞凋亡增强，可能是通过 p53 突变结直肠癌细胞中的 ATM/Chk/Cdc25C/Cdc2 途径
DOI：
10.3892/ijo.2018.4514
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发表时间：
2018
影响因子：
5.2
通讯作者：
Zhang Zhen
中科院分区：
文献类型：
--
作者：
Wang Yaqi;Yang Lifeng;Zhang Jing;Zhou Menglong;Shen Lijun;Deng Weijuan;Liang Liping;Hu Ran;Yang Wang;Yao Ye;Zhang Hui;Zhang Zhen
