Id1 is downregulated by hepatocyte growth factor via ERK-dependent and -independent signaling pathways, leading to increased expression of p16INK4a in hepatoma cells.
Id1 is downregulated by hepatocyte growth factor via ERK-dependent and -independent signaling pathways, leading to increased expression of p16INK4a in hepatoma cells.
复制标题
肝细胞生长因子通过 ERK 依赖性和非依赖性信号通路下调 Id1，导致肝癌细胞中 p16INK4a 的表达增加。
DOI：
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发表时间：
2009
期刊：
影响因子：
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通讯作者：
T. Tanaka^*
中科院分区：
文献类型：
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作者：
K. Ushio T. Hashimoto;N. Kitamura;T. Tanaka^*
