Source-Specific Social Support and Circulating Inflammatory Markers Among White-Collar Employees
Source-Specific Social Support and Circulating Inflammatory Markers Among White-Collar Employees
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DOI：
10.1007/s12160-013-9555-x
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发表时间：
2014-06-01
影响因子：
3.8
通讯作者：
Takahashi, Masaya
中科院分区：
文献类型：
--
作者：
Nakata, Akinori;Irie, Masahiro;Takahashi, Masaya
尽管社会支持对心血管健康有积极作用，但支持来源(主管、同事、家人/朋友)影响炎症标志物的途径尚不完全清楚。我们调查了社会支持与炎症标志物之间的独立和调节关系。对137名男性白领员工进行了抽血检测超敏C反应蛋白(hs-CRP)、白介素6(IL-6)、肿瘤坏死因子α(TNF-α)、单核细胞和白细胞计数，控制协变量的多元线性回归分析显示，主管支持与IL-6呈负相关(β=-0.24，p<；而同事支持与肿瘤坏死因子-α有微弱的相关性(β=-0.16，p<；0.10)。来自家人/朋友的支持与炎症性标志物无关。来自直属上司的社会支持可能是一种潜在的机制，通过社会支持对职场男性的炎症性标志物产生有利影响。
Despite known beneficial effects of social support on cardiovascular health, the pathway through which sources of support (supervisor, coworkers, family/friends) influence inflammatory markers is not completely understood.We investigated the independent and moderating associations between social support and inflammatory markers.A total of 137 male white-collar employees underwent a blood draw for measurement of high-sensitive C-reactive protein (hs-CRP), interleukin-6 (IL-6), tumor necrosis factor alpha (TNF-alpha), monocyte and leukocyte counts, and completed a questionnaire on social support.Multivariable linear regression analyses controlling for covariates revealed that supervisor support was inversely associated with IL-6 (beta = -0.24, p < 0.01) while coworker support was marginally associated with TNF-alpha (beta = -0.16, p < 0.10). Support from family/friends was not associated with inflammatory markers.Social support from the immediate supervisor may be a potential mechanism through which social support exerts beneficial effects on inflammatory markers in working men.