Cardiac output during sleep at altitude.
Cardiac output during sleep at altitude.
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在海拔高度睡眠期间的心输出量。
DOI：
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发表时间：
1977
期刊：
影响因子：
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通讯作者：
Horvath Sm
中科院分区：
文献类型：
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作者：
Miller Jc;Horvath Sm
：采用阻抗心动图研究了两名男性和两名女性受试者的低压缺氧和睡眠（通过EEG评估）对心输出量（Q）的影响。三个晚上在746度（152米），然后两个晚上在493度（3500米），所有在低压舱。除了急性高原病（AMS）外，观察到相对正常的睡眠。同步睡眠的正常水平的发生意味着睡眠换气不足发生。在大约0500时观察到Q的最低点，这意味着O2输送减少。有人建议，低压缺氧，睡眠换气不足，减少O2输送可能相互作用，产生AMS。
: The effects of hypobaric hypoxia and sleep, assessed by EEG, upon cardiac output (Q) were studied by impedance cardiography using two male and two female subjects. Three nights were spent at 746 torr (152 m) followed by two nights at 493 torr (3500 m), all in a hypobaric chamber. Relatively normal sleep was observed except in the presence of Acute Mountain Sickness (AMS). The occurrence of normal levels of synchronized sleep implied that sleep hypoventilation occurred. A nadir of Q was observed at about 0500 hours, implying reduced O2 delivery. It was suggested that hypobaric hypoxia, sleep hypoventilation, and reduced O2 delivery may have interacted to produce AMS.