Influence of contralateral noise on distortion product latency in humans: Is the medial olivocochlear efferent system involved?
Influence of contralateral noise on distortion product latency in humans: Is the medial olivocochlear efferent system involved?
复制标题
DOI：
10.1121/1.419635
复制
发表时间：
1997-10-01
影响因子：
2.4
通讯作者：
CheryCroze, S
中科院分区：
文献类型：
--
作者：
Giraud, AL;Wable, J;CheryCroze, S
为了检验内侧传出神经被激活时耳蜗反应的时间改变的假设，在存在和不存在对侧宽带噪声的情况下，使用2f 1-f2失真产物的相位测量来估计16名正常受试者的耳声发射潜伏期（组潜伏期方法）。在较低的频率（0.8-2.7 kHz）发射的潜伏期显着减少，发现在存在的对侧声音的水平增加，这种影响消失时，主音水平增加到60 dB SPL。为了确保效果不归因于涉及中耳结构的机制，容易受到对侧声音的激活，在前庭神经切断术期间传出神经被切断的7名受试者和2名镫骨肌麻痹的受试者中进行了潜伏期测量。患者的结果进行了比较，在三个完整的传出束手术对照患者，并在其他八个正常人。所有受试者组均表现出对侧声潜伏期缩短，但传出神经系统切断的患者潜伏期延长。(C)1997年美国声学学会。[S 0001 -4966（97）02909-3]PACS编号：43.64.Jb、43.64.Wn [RDF]。
To test the hypothesis of temporal modifications of cochlear responses when medial efferents are activated, otoacoustic emission latencies were estimated in 16 normal human subjects, in the presence and absence of a contralateral broadband noise, using measurements of the phase of the 2f1-f2 distortion product (group latency method). Significant decrease in the latency of lower frequency (0.8-2.7 kHz) emissions was found in the presence of increasing levels of contralateral sound, and this effect disappeared when the primary-tone levels increased to 60 dB SPL. To ensure that effects were not attributable to mechanisms involving middle ear structures, susceptible to activation by contralateral sound, latency measures were performed in seven subjects whose efferents were severed during a vestibular neurotomy and in two subjects with paralyzed stapedial muscle. Results in patients were compared to those obtained in three surgical control patients with intact efferent bundle, and in eight other normal subjects. All the subject groups exhibited a decrease in latency under contralateral sound except the patients with the severed efferent system who showed increased latencies. (C) 1997 Acoustical Society of America. [S0001-4966(97)02909-3]PACS numbers: 43.64.Jb, 43.64.Wn [RDF].