Myocardial contraction band lesions in patients with fatal asthma: possible neurocardiologic mechanisms.
Myocardial contraction band lesions in patients with fatal asthma: possible neurocardiologic mechanisms.
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致命性哮喘患者的心肌收缩带病变：可能的神经心脏病学机制。
DOI：
10.1164/arrd.1987.135.2.498
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发表时间：
1987
期刊：
影响因子：
--
通讯作者：
Strunk,RC
中科院分区：
文献类型：
--
作者：
Drislane,FW;Samuels,MA;Kozakewich,H;Schoen,FJ;Strunk,RC
心肌收缩带坏死(MCBN)发生于儿茶酚胺输注、中枢神经系统刺激、应激和短暂性心肌缺血再灌流。在13例死于哮喘的儿童中，有4例出现了MCBN，这表明这种心脏损害可能导致了一些哮喘患者的死亡。4例MCBN患者中有2例未经静脉或心内途径接受交感神经治疗。因此，除了大剂量输注儿茶酚胺外，其他机制可能与哮喘患者心脏损害的产生有关。
Myocardial contraction band necrosis (MCBN) occurs in catecholamine infusion, central nervous system stimulation, stress, and transient myocardial ischemia with reperfusion. In 4 of 13 children who died with asthma, MCBN was present, suggesting that this cardiac lesion may contribute to the deaths of some asthmatic patients. Two of the 4 patients who had MCBN had not received sympathomimetics intravenously or by an intracardiac route. Therefore, mechanisms other than infusions of large doses of catecholamines are probably involved in production of this cardiac lesion in asthmatics.