Sleep patterns and obesity in childhood.
Sleep patterns and obesity in childhood.
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DOI：
10.1097/med.0000000000000125
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发表时间：
2015-02
期刊：
影响因子：
--
通讯作者：
LeBourgeois MK
中科院分区：
文献类型：
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作者：
Miller AL;Lumeng JC;LeBourgeois MK
以突出最近关于儿童睡眠肥胖相关性的研究结果。我们将重点放在睡眠持续时间、睡眠时间和时型上，并描述潜在的睡眠肥胖关联机制。睡眠不佳在儿童中越来越普遍，儿童早期睡眠时间短与肥胖之间的联系一直被发现。对婴儿期的了解较少，对青少年的研究结果也不一致。睡眠时间模式也可能导致肥胖风险。最近，可变和改变的睡眠时间和晚间时型与成年人的肥胖有关；对儿童的了解较少。此外，人们对联合机制的了解也很少。进食时间、饮食摄入量、肥胖饮食行为和食欲调节激素的变化已被确定为睡眠肥胖症关联的可能机制，并可能成为未来研究的前景。需要对儿童进行纵向和实验性的工作，以确定关联的性质。除了睡眠时间外，睡眠时间模式可能会导致肥胖风险。生物和行为过程被认为是解释这种联系的机制。了解不良睡眠模式可能增加儿童肥胖风险的途径，可能会为干预提供新的途径。
To highlight the recent findings on sleep–obesity associations in children. We focus on sleep duration, sleep timing and chronotype, and describe the potential mechanisms underlying sleep–obesity associations. Poor sleep is increasingly common in children and associations between short sleep duration in early childhood and obesity are consistently found. Less is known about the infancy period, and the findings in adolescents are inconsistent. Sleep timing patterns may also contribute to obesity risk. Variable and shifted sleep schedules and evening chronotypes have recently been linked to adiposity in adults; less is known about children. Further, there is little understanding regarding the mechanisms of association. The timing of eating, dietary intake, obesogenic eating behaviors, and changes in appetite-regulating hormones have been identified as possible mechanisms for sleep–obesity associations and may be promising avenues for future research. Longitudinal and experimental work with children is needed to determine the nature of associations. Beyond sleep duration, sleep timing patterns may contribute to obesity risk. Biological and behavioral processes have been proposed as mechanisms that may explain the association. Understanding the pathways through which poor sleep patterns could increase obesity risk in children may provide novel avenues for intervention.