Respiratory biofeedback-assisted therapy in panic disorder
Respiratory biofeedback-assisted therapy in panic disorder
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DOI：
10.1177/0145445501254006
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发表时间：
2001-09-01
影响因子：
2.3
通讯作者：
Roth, WT
中科院分区：
文献类型：
--
作者：
Meuret, AE;Wilhelm, FH;Roth, WT
作者描述了一种新的方法学改进的行为治疗恐慌患者使用呼吸生物反馈从手持二氧化碳测定设备。治疗原理是基于这样的假设，即过度换气导致的持续性低碳酸血症是产生和维持恐慌的关键机制。短暂的4周生物反馈治疗旨在通过患者环境中的呼吸练习，自愿增加自我监测的呼气末二氧化碳分压（PCO 2），并降低呼吸频率和不稳定性。4名患者的初步结果表明，该疗法成功地减少了恐慌症状和与恐慌症相关的其他心理特征。从家庭训练、治疗前和治疗后24小时动态监测以及随访时的实验室评估中获得的生理数据表明，患者开始时的静息PCO 2水平较低，治疗期间这些水平升高，并在治疗后和/或随访时保持这些水平。PCO 2和呼吸频率之间的部分分离质疑呼吸频率是否应该是惊恐障碍呼吸训练的主要焦点。
The authors describe a new methodologically improved behavioral treatment for panic patients using respiratory biofeedback from a handheld capnometry device. The treatment rationale is based on the assumption that sustained hypocapnia resulting from hyperventilation is a key mechanism in the production and maintenance of panic. The brief 4-week biofeedback therapy is aimed at voluntarily increasing self-monitored end-tidal partial pressure of carbon dioxide (PCO2) and reducing respiratory rate and instability through breathing exercises in patients' environment. Preliminary results from 4 patients indicate that the therapy was successful in reducing panic symptoms and other psychological characteristics associated with panic disorder. Physiological data obtained from home training, 24-hour ambulatory monitoring pretherapy and posttherapy, and laboratory assessment at follow-up indicate that patients started out with low resting PCO2 levels, increased those levels during therapy, and maintained those levels at posttherapy and/or follow-up. Partial dissociation between PCO2 and respiratory rate questions whether respiratory rate should be the main focus of breathing training in panic disorder.