PLASMA NOREPINEPHRINE AND HEART-RATE DYNAMICS DURING RECOVERY FROM SUBMAXIMAL EXERCISE IN MAN
PLASMA NOREPINEPHRINE AND HEART-RATE DYNAMICS DURING RECOVERY FROM SUBMAXIMAL EXERCISE IN MAN
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DOI：
10.1007/bf02332222
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发表时间：
1989-07-01
影响因子：
3
通讯作者：
VEICSTEINAS, A
中科院分区：
文献类型：
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作者：
PERINI, R;ORIZIO, C;VEICSTEINAS, A
心率 (HR) 和静脉血去甲肾上腺素浓度 [NE]（作为交感神经活动 (SNA) 的表达）的时间过程，在 6 名久坐的年轻人中进行了研究，他们在 21% .+- 的自行车测力计运动的三个阶段恢复期间进行了研究。 2.8%, 43%.+-. 2.1% 和 65% .+-。分别为 .ovrhdot.VO2max 的 2.3%（平均值 .+-.SE）。 HR随着.tau呈单指数下降。值为 13.6 .+-。 1.6 秒，32.7 .+-。 5.6 秒和 55.8 .+-。三个运动时段分别为8.1s。在低运动水平下，[NE] 没有发现变化。在中等和高运动强度下：（a）[NE]在运动第5分钟显着增加（Δ[NE]分别= 207.7 .+-. 22.5 pg .cntdot. ml-1和521.3 .+-. 58.3 pg .cntdot. ml-1）； (b) 1 分钟滞后后 [NE] 呈指数下降（τ 分别 = 87 秒和 101 秒）； (c) 在第 1 分钟 HR 减少约 35 次.cntdot。分钟-1； (d) 从恢复的第 2 分钟到第 5 分钟，HR 和 [NE] 呈线性相关（100 pg .cntdot. ml-1 .DELTA.[NE] .simeq. 5 次心跳 .cntdot. min-1）。在恢复的第一分钟，无论运动强度如何，心率的调整似乎主要是由于迷走神经张力的迅速恢复。 HR 向静息值的进一步下降可归因于 SNA 恢复至运动前水平。
The time course of heart rate (HR) and venous blood norepinephrine concentration [NE], as an expression of the sympathetic nervous activity (SNA), was studied in six sedentary young men during recovery from three periods of cycle ergometer exercise at 21% .+-. 2.8%, 43% .+-. 2.1% and 65% .+-. 2.3% of .ovrhdot.VO2max respectively (mean .+-. SE). The HR decreased mono-exponentially with .tau. values of 13.6 .+-. 1.6 s, 32.7 .+-. 5.6 s and 55.8 .+-. 8.1 s respectively in the three periods of exercise. At the low exercise level no change in [NE] was found. At medium and high exercise intensity: (a) [NE] increased significantly at the 5th min of exercise (.DELTA.[NE] = 207.7 .+-. 22.5 pg .cntdot. ml-1 and 521.3 .+-. 58.3 pg .cntdot. ml-1 respectively); (b) after a time lag of 1 min [NE] decreased exponentially (.tau. = 87 s and 101 s respectively); (c) in the 1st min HR decreased about 35 beats .cntdot. min-1; (d) from the 2nd to 5th min of recovery HR and [NE] were linearly related (100 pg .cntdot. ml-1 .DELTA.[NE] .simeq. 5 beats .cntdot. min-1). In the 1st min of recovery, independent of the exercise intensity, the adjustment of HR appears to have been due mainly to the prompt restoration of vagal tone. The further decrease in HR toward the resting value could then be attributed to the return of SNA to the pre-exercise level.