GLAUCOMA SECONDARY TO ELEVATED EPISCLERAL VENOUS-PRESSURE
GLAUCOMA SECONDARY TO ELEVATED EPISCLERAL VENOUS-PRESSURE
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DOI：
10.1055/s-2008-1050284
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发表时间：
1988-11-01
影响因子：
0.8
通讯作者：
GUTHOFF, R
中科院分区：
文献类型：
--
作者：
JORGENSEN, JS;GUTHOFF, R
1983年至1987年，作者研究了64例巩膜外血管扩张和开角型青光眼患者。巩膜上静脉压升高是导致眼内压升高的原因。临床上，引起静脉压升高的原因包括自发性颈动脉海绵窦瘘、Sturge-Weber综合征、眼眶肿瘤、内分泌突眼、前巩膜炎和特发性病例。本文发现造成巩膜上静脉压升高的各种病理生理机制，并讨论其临床过程及并发症。
Between 1983 and 1987 the authors studied 64 patients with dilated episcleral vessels and open-angle glaucoma. Elevated episcleral venous pressure was found to be the cause of the increased intraocular pressure. Clinically, the cause of elevation of venous pressure included spontaneous carotid cavernous fistulas, Sturge-Weber syndrome, orbital tumors, endocrine exophthalmos, anterior scleritis and idiopathic cases. A variety of pathophysiological mechanisms causing elevated episcleral venous pressure were found. The clinical course and complications associated with elevated episcleral venous pressure are discussed.