A new way to regulate inflammation: selective autophagic degradation of IKKγ mediated by ANGPTL8
A new way to regulate inflammation: selective autophagic degradation of IKKγ mediated by ANGPTL8
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调节炎症的新途径：ANGPTL8介导的IKKγ选择性自噬降解
DOI：
10.15698/cst2018.03.128
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发表时间：
2018-02
期刊：
影响因子：
6.4
通讯作者：
Huang Kun
中科院分区：
文献类型：
--
作者：
Zhang Yu;Zheng Ling;Huang Kun
转录因子核因子-κB（NF-κB）在炎症和免疫调节中起关键作用。然而，NF-κB激活的确切机制尚未完全阐明。我们最近发现血管生成素样蛋白8（Angiopoietin-like protein 8，ANGPTL 8，als
Transcription factor nuclear factor-κB (NF-κB) plays pivotal roles in the regulation of inflammation and immunity. However, the precise mechanism of NF-κB activation is not fully elucidated. We recently found that Angiopoietin like protein 8 (ANGPTL8, als