HMGB1 release by H2O2-induced hepatocytes is regulated through calcium overload and 58-F interference

HMGB1 release by H2O2-induced hepatocytes is regulated through calcium overload and 58-F interference
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H2O2 诱导的肝细胞释放 HMGB1 通过钙超载和 58-F 干扰进行调节

DOI:
10.1038/cddiscovery.2017.8
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发表时间:
2017-04
影响因子:
7
通讯作者:
Xiaoling Wang
Xiaoling Wang
中科院分区:
医学2区
文献类型:
--
作者:
Pei Zhao;Tingjie Ye;Xiaofen Yan;Xudong Hu;Ping Liu;Xiaoling Wang

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HMGB1由受伤或死亡的细胞被动释放,并加剧炎症过程。HMGB1的释放和钙超载均被报道为h2o2诱导损伤的重要介质。然而,两者之间的潜在联系
HMGB1 is passively released by injured or dying cells and aggravates inflammatory processes. The release of HMGB1 and calcium overload have each been reported to be important mediators of H2O2-induced injury. However, a potential connection between these
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