THE HYPOKALEMIA OF ACUTE MYOCARDIAL-INFARCTION
THE HYPOKALEMIA OF ACUTE MYOCARDIAL-INFARCTION
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DOI：
10.1177/000456328602300211
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发表时间：
1986-03-01
影响因子：
2.2
通讯作者：
REID, W
中科院分区：
文献类型：
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作者：
RODGER, JC;SIMPSON, E;REID, W
在一项回顾性研究中，我们观察到低钾血症在心肌梗死急性期很常见，它与严重的室性心律失常和高死亡率有关，不能仅仅归因于利尿剂治疗。我们认为，这是由于细胞内钾的转移，可能是在响应增加的儿茶酚胺分泌。“公认肾上腺髓质儿茶酚胺在钾稳态中起作用”：当将肾上腺素输注到正常受试者体内时，在输注开始后30分钟内发生低钾血症，并在停止输注后1-2小时内恢复正常。因此，这种低钾血症被描述为“短暂的”，就像这种实验性诱导的低钾血症一样，
In a retrospective study, we observed that hypokalaemia was common in the acute phase of myocardial infarction, that it was associated with serious ventricular arrhythmias and a high mortality, and that it could not be attributed solely to diuretic therapy. We suggested that it was due to an intracellular shift of potassium, possibly in response to increased catecholamine secretion.'It is accepted that adrenal medullary catecholamines have a role in potassium homeostasis": when adrenaline is infused into normal subjects, the resulting hypokalaemia occurs within 30 min of the commencement of the infusion and returns to normal over 1-2 h following cessation of the infusion. This hypokalaemia has thus been described as' transient'v/Like this experimentally-induced hypoka-