Increase of endosomal Sec61 enhances antigen cross-presentation and induces immune glomerular injury: a novel ‘self-organized criticality theory’ explaining the cause of systemic lupus erythematosus.
Increase of endosomal Sec61 enhances antigen cross-presentation and induces immune glomerular injury: a novel ‘self-organized criticality theory’ explaining the cause of systemic lupus erythematosus.
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内体 Sec61 的增加增强抗原交叉呈递并诱导免疫肾小球损伤：一种新的“自组织临界理论”解释了系统性红斑狼疮的病因。
DOI：
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发表时间：
2015
期刊：
影响因子：
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通讯作者：
Shiozawa S.
中科院分区：
文献类型：
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作者：
Tsumiyama K;Shiozawa S.
