Limits to a left hemisphere explanation for specific language impairment.
Limits to a left hemisphere explanation for specific language impairment.
复制标题
对特定语言障碍的左半球解释的限制。
DOI：
10.1044/jshr.3704.824
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发表时间：
1994
期刊：
影响因子：
--
通讯作者：
Eisele,JA
中科院分区：
文献类型：
--
作者：
Aram,DM;Eisele,JA
单侧左半球损伤作为特定语言障碍（SLI）的神经基础的解释模型的假设似乎并不足以为大多数儿童与SLI。单侧脑损伤的儿童在各种语言测量中的功能明显低于神经功能完好的同龄人，但很少有缺陷像SLI中看到的那样持久或严重。然而，至少在两种情况下，儿童单侧病变后的语言障碍与某些类型的SLI相似。第一种发生在前部灰质和白色物质结构的皮质下损伤之后，通常会导致明显的语言和学习障碍。第二个相似之处在于，语言“延迟”儿童和已知单侧病变儿童的发展过程相似，即语言的开始和发展在学龄前阶段缓慢，但到学龄时正常化，长期语言学习缺陷最小。
The hypothesis of unilateral left hemisphere damage as an explanatory model for the neurological basis of specific language impairment (SLI) does not appear to be sufficient for most children with SLI. Children with unilateral brain lesions have been shown to function significantly lower than their neurologically intact peers on a variety of language measures, yet few of the deficits noted are as persistent or severe as those seen in SLI. In at least two instances, however, language symptomatology following unilateral lesions in children does parallel some types of SLI. The first occurs following subcortical damage to anterior grey and white matter structures that typically results in pronounced language and learning disorders. The second parallel lies in the similar developmental course shared by children with “delayed” language and children with known unilateral lesions, whereby language onset and development is slow in the preschool years but normalizes by school age, with minimal long-term language-learning deficits.