Modelling spatial contrast sensitivity functions for chromatic and luminance-modulated gratings
Modelling spatial contrast sensitivity functions for chromatic and luminance-modulated gratings
复制标题
DOI：
10.1016/s0042-6989(98)00273-9
复制
发表时间：
1999-06
期刊：
影响因子：
1.8
通讯作者：
J. Rovamo;Mia I. Kankaanpää;H. Kukkonen
中科院分区：
文献类型：
--
作者：
J. Rovamo;Mia I. Kankaanpää;H. Kukkonen
我们扩展了我们的非彩色空间视觉的检测模型（Rovamo，J.，Mustonen，J.，& Näsänen，R.（1994年a）。对比敏感度作为视网膜照度和光栅面积的函数的建模。Vision Research，34，1301 - 1314）通过考虑到以下事实而将等照度色光栅与色觉联系起来：由于视网膜神经节细胞和背外侧膝状体核（dLGN）神经元的空间色性，等照度色光栅不受皮质前侧抑制的影响。然后，我们使用马伦的实验数据（马伦，K. J.（1985年）。人类色觉对红绿蓝黄色光栅的对比敏感度。Journal of Physiology，359，381 - 400）。亮度调制的绿色和黄色光栅的空间对比敏感度函数的带通形状转换为低通形状，类似于当通过将亮度对比敏感度除以空间频率（即，除以af，其中a = 1 °）来计算去除皮质前侧抑制对光栅对比度的影响时，红-绿色和蓝-黄色等亮度光栅的彩色空间对比敏感度函数。在去除侧抑制的直接影响后，色光栅和亮度光栅的空间对比敏感度函数之间仍然存在残余形状差异。这是由于经皮质前区侧抑制高通滤波后的量子噪声间接降低了光栅的可见度。当这种量子噪声的间接影响也被删除时，亮度光栅的对比灵敏度在所有空间频率下大约是彩色光栅灵敏度的两倍。这显然是由于对手阶段的等光光栅的色彩对比度（科尔，G. R.，Hine，T. & McIihagga，W.（1993年）。L-、M-和S-视锥对比度空间中的检测机制。Journal of the Optical Society of America A，10，38 - 51）的亮度对比度约为其彩色分量中任一个的亮度对比度的一半。因此，如果等照度彩色光栅的对比度不表示为一个彩色分量光栅相对于其自身背景的迈克尔逊对比度（马伦，K. J.（1985年）。人类色觉对红绿蓝黄色光栅的对比敏感度。Journal of Physiology，359，381 - 400），但是作为在对立阶段的色彩对比度，对比灵敏度对于色彩和亮度光栅将是相同的。
We extended our detection model of achromatic spatial vision (Rovamo, J., Mustonen, J., & Näsänen, R. (1994a). Modelling contrast sensitivity as a function of retinal illuminance and grating area. Vision Research, 34, 1301–1314) to colour vision by taking into account the fact that due to the spatio-chromatic opponency of retinal ganglion cells and dorsal lateral geniculate nucleus (dLGN) neurons, equiluminous chromatic gratings are not affected by precortical lateral inhibition. We then tested the extended model by using Mullen’s experimental data (Mullen, K. J. (1985). The contrast sensitivity of human color vision to red–green and blue–yellow chromatic gratings. Journal of Physiology, 359, 381–400). The band-pass shape of the spatial contrast sensitivity function for luminance-modulated green and yellow gratings transformed to a low-pass shape, resembling the chromatic spatial contrast sensitivity function for red–green and blue–yellow equiluminous gratings, when the effect of precortical lateral inhibition on grating contrast was computationally removed by dividing luminance contrast sensitivities by spatial frequency (i.e. by af, where a=1°). After the removal of this direct effect of lateral inhibition, there still remained a residual shape difference between the spatial contrast sensitivity functions for chromatic and luminance gratings. It was due to indirect reduction of grating visibility by quantal noise high-pass filtered by precortical lateral inhibition. When this indirect effect of quantal noise was also removed, contrast sensitivity for luminance gratings was about twice the sensitivity for chromatic gratings at all spatial frequencies. This was evidently due to the fact that the chromatic contrast of the equiluminous grating at the opponent stage (Cole, G. R., Hine, T. & McIihagga, W. (1993). Detection mechanisms in L-, M-, and S-cone contrast space. Journal of the Optical Society of America A, 10, 38–51) was about half of the luminance contrast of either of its chromatic component. Thus, if the contrast of the equiluminous chromatic grating were not expressed as the Michelson contrast of one chromatic component grating against its own background (Mullen, K. J. (1985). The contrast sensitivity of human color vision to red–green and blue–yellow chromatic gratings. Journal of Physiology, 359, 381–400) but as chromatic contrast at the opponent stage, contrast sensitivity would be the same for chromatic and luminance gratings.