Correction to: 'Anastasis: recovery from the brink of cell death'.

Correction to: 'Anastasis: recovery from the brink of cell death'.
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DOI:
10.1098/rsos.181629
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发表时间:
2018-10
影响因子:
3.5
通讯作者:
Tang HL
Tang HL
中科院分区:
综合性期刊3区
文献类型:
--
作者:
Tang HM;Tang HL

文献摘要

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表1.细胞死亡事件的恢复。细胞死亡事件涉及磷脂酰丝氨酸的外化(体外)[21-24]磷脂酰丝氨酸的外化(体内)[25]细胞色素c释放[26]不完全MOMP[27]线粒体片段化[21,26,28,29]半胱天冬酶激活(体外)[21,26,28-33]半胱天冬酶活化(体内)[34,35]质膜起泡[21,26,28,29]细胞收缩[21,24,26,28-30,32] DNA损伤[21,27,31]核凝聚[21,26,28,29,32] RIPK 3激活[24,36]凋亡小体形成和细胞碎裂[26]在第3.2节第四段,第三句应改为:“值得注意的是,培养的细胞已被证明可以耐受亚致死性半胱天冬酶活性而不触发凋亡[31,128 -130]。”在第3.2节,第四段,倒数第三句应改为:“转录在anastasis期间也是活跃的[21,29,32],并且可以为恢复细胞修复损伤并恢复正常形态产生构建模块。在第3.3节第三段,第一句应改为:“iMOMP和半胱天冬酶-3的亚致死激活也可促进DNA损伤和致癌转化,推测是通过凋亡DNA酶,而
Table 1. Reversal of cell death events. cell death events references externalization of phosphatidylserine (in vitro)[21–24] externalization of phosphatidylserine (in vivo)[25] cytochrome c release[26] incomplete MOMP[27] mitochondrial fragmentation[21, 26, 28, 29] caspase activation (in vitro)[21, 26, 28–33] caspase activation (in vivo)[34, 35] plasma membrane blebbing[21, 26, 28, 29] cell shrinkage[21, 24, 26, 28–30, 32] DNA damage[21, 27, 31] nuclear condensation[21, 26, 28, 29, 32] RIPK3 activation[24, 36] apoptotic body formation and cell fragmentation [26]In Section 3.2, fourth paragraph, third sentence should read:‘Remarkably, cultured cells have been shown to tolerate sublethal caspase activity without triggering apoptosis [31,128–130].’In Section 3.2, fourth paragraph, third last sentence should read:‘Transcription is also active during anastasis [21, 29, 32], and that can generate building blocks for the recovering cells to repair damage and regain normal morphology.’In Section 3.3, third paragraph, first sentence should read:‘DNA damage and oncogenic transformation can also be promoted by iMOMP and sublethal activation of caspase-3, presumably through apoptotic DNases, whereas