Store-operated Ca2+ Entry (SOCE) Plays a Role in the Polarization of Neutrophil-like HL-60 Cells by Regulating the Activation of Akt, Src, and Rho Family GTPases
Store-operated Ca2+ Entry (SOCE) Plays a Role in the Polarization of Neutrophil-like HL-60 Cells by Regulating the Activation of Akt, Src, and Rho Family GTPases
复制标题
钙池操纵的 Ca2 进入 (SOCE) 通过调节 Akt、Src 和 Rho 家族 GTP 酶的激活在中性粒细胞样 HL-60 细胞的极化中发挥作用
DOI：
10.1159/000339059
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发表时间：
2012-06
影响因子：
--
通讯作者：
Zou, Fei
中科院分区：
文献类型：
--
作者：
Zou, Wenying;Meng, Xiaojing;Cai, Chunqing;Zou, Mengchen;Tang, Shihao;Chu, Xinwei;Wang, Xubu;Zou, Fei
神经元极化是参与先天性免疫应答的基本活动，并且其可以由细胞外Ca 2+内流启动，该过程主要通过钙库操纵的Ca 2+内流（SOCE）介导。然而，SOCE参与的机制
Neutrophil polarization is a basic activity involved in the innate immune response, and it may be initiated by extracellular Ca2+ entry, a process primarily mediated through store-operated Ca2+ entry (SOCE). Yet, the mechanisms by which SOCE participates