Kinesthetic Alexia due to Left Parietal Lobe Lesions
Kinesthetic Alexia due to Left Parietal Lobe Lesions
复制标题
左顶叶病变导致的动觉失读症
作者：
N. Ihori;M. Kawamura;S. Araki;J. Kawachi
为了探讨动觉性失读症的神经心理学机制，我们要求7例左顶叶损伤后表现为动觉性失读症并保留视觉阅读的患者执行包括动觉性书写复制（写下与动觉刺激相同的字母）、动觉性朗读、视觉书写复制（抄写字母）和视觉朗读平假名（日语唱音）的任务。我们比较了每位患者在这些任务中的表现和病变部位。结果表明，以下任何一种功能的缺陷都可能导致动觉性失读症：(1)从动觉刺激中检索文字的动觉图像（运动印痕）；(2)动觉图像本身；(3)从动觉图像中获取跨模态关联；(4)跨模态关联本身（从动觉图像中检索听觉和视觉图像）。这些因素似乎都与左顶叶的不同病变部位有关。
To investigate the neuropsychological mechanisms of kinesthetic alexia, we asked 7 patients who showed kinesthetic alexia with preserved visual reading after damage to the left parietal region to perform tasks consisting of kinesthetic written reproduction (writing down the same letter as the kinesthetic stimulus), kinesthetic reading aloud, visual written reproduction (copying letters), and visual reading aloud of hiragana (Japanese phonograms). We compared the performance in these tasks and the lesion sites in each patient. The results suggested that deficits in any one of the following functions might cause kinesthetic alexia: (1) the retrieval of kinesthetic images (motor engrams) of characters from kinesthetic stimuli, (2) kinesthetic images themselves, (3) access to cross-modal association from kinesthetic images, and (4) cross-modal association itself (retrieval of auditory and visual images from kinesthetic images of characters). Each of these factors seemed to be related to different lesion sites in the left parietal lobe.
影响因子：
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作者：
ALEXANDER, MP;FISCHER, RS;FRIEDMAN, R
通讯作者：
FRIEDMAN, R