Deoxycytidine kinase participates in the regulation of radiation-induced autophagy and apoptosis in breast cancer cells.
Deoxycytidine kinase participates in the regulation of radiation-induced autophagy and apoptosis in breast cancer cells.
复制标题
脱氧胞苷激酶参与乳腺癌细胞辐射诱导的自噬和凋亡的调节。
DOI：
10.3892/ijo.2018.4250
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发表时间：
2018-03
期刊：
影响因子：
--
通讯作者：
Liu X
中科院分区：
文献类型：
--
作者：
Zhong R;Liang B;Xin R;Zhu X;Liu Z;Chen Q;Hou Y;Jin Z;Qi M;Ma S;Liu X
脱氧胞苷激酶（dCK）是一种限速酶，对内源性脱氧核苷的磷酸化和许多核苷类似物的抗肿瘤活性至关重要。dCK响应于电离辐射（IR）而被激活，
Deoxycytidine kinase (dCK) is a rate limiting enzyme critical for the phosphorylation of endogenous deoxynucleosides and for the anti‑tumor activity of many nucleoside analogs. dCK is activated in response to ionizing radiation (IR) and it is required for