Hans Berger (1873-1941), Richard Caton (1842-1926), and electroencephalography.
Hans Berger (1873-1941), Richard Caton (1842-1926), and electroencephalography.
复制标题
DOI：
10.1136/jnnp.74.1.9
复制
发表时间：
2003-01-01
期刊：
影响因子：
--
通讯作者：
Haas, L F
中科院分区：
文献类型：
--
作者：
Haas, L F
自发性视网膜静脉搏动被认为是视网膜静脉穿过视盘时口径的细微变化。静脉搏动背后的物理原理一直是争论的焦点。最初的理论认为，脉动的发生是因为眼内压随着脉搏压的升高而升高。这篇文章提出了一个论点，认为情况并非如此。脉动实际上是由当视网膜静脉穿过筛板时沿视网膜静脉沿着的压力梯度的变化引起的。压力梯度由于眼内空间和脑脊液之间的脉压差而变化。这一点的重要性在于，随着颅内压升高，颅内脉压升高至等于眼内脉压，自发性静脉搏动停止。因此，自发性静脉脉动的停止是颅内压升高的敏感标志。本文讨论了自发性静脉搏动缺失的特异性，并描述了如何对患者进行检查以最好地引出这一重要体征。
Spontaneous retinal venous pulsation is seen as a subtle variation in the calibre of the retinal vein (s) as they cross the optic disc. The physical principles behind the venous pulsations has been the point of much debate. Initial theories suggested that the pulsation occurred because of the rise in intraocular pressure in the eye with the pulse pressure. This article presents an argument that this is not the case. The pulsations are in fact caused by variation in the pressure gradient along the retinal vein as it traverses the lamina cribrosa. The pressure gradient varies because of the difference in the pulse pressure between the intraocular space and the cerebrospinal fluid. The importance of this is that as the intracranial pressure rises the intracranial pulse pressure rises to equal the intraocular pulse pressure and the spontaneous venous pulsations cease. Thus it is shown that cessation of the spontaneous venous pulsation is a sensitive marker of raised intracranial pressure. The article discusses the specificity of the absence of spontaneous venous pulsation and describes how the patient should be examined to best elicit this important sign.