In vivo kinematics of the tibiotalar joint after lateral ankle instability.
In vivo kinematics of the tibiotalar joint after lateral ankle instability.
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DOI：
10.1177/0363546509337578
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发表时间：
2009-11
期刊：
影响因子：
--
通讯作者：
DeFrate LE
中科院分区：
文献类型：
--
作者：
Caputo AM;Lee JY;Spritzer CE;Easley ME;DeOrio JK;Nunley JA 2nd;DeFrate LE
先前的研究表明，距腓前韧带的损伤可能与踝关节运动学的改变和骨关节炎的发展有关。然而，ATFL 损伤对踝关节体内运动学的影响尚不清楚。根据 ATFL 纤维的方向，我们假设 ATFL 缺陷会导致距骨相对于胫骨的前平移增加和内旋增加。受控实验室研究。对 9 名单侧 ATFL 损伤患者踏上水平表面时的脚踝进行了比较。运动学测量是作为增加负载的函数进行的。使用磁共振成像和正交透视，在同一个体的 ATFL 缺陷和完整踝关节中测量了胫距关节的体内运动学。与完整的对侧对照相比，ATFL 缺陷踝关节的距骨内旋、前平移和上平移具有统计学显着性增加。例如，在 100% 体重时，ATFL 缺陷的脚踝表现出统计上显着的前移增加 0.9 ± 0.5 毫米 (p = 0.008)。在 100% 体重时，ATFL 缺陷踝关节相对于完整踝关节内旋 5.7 ± 3.6° (p = 0.008)。在 100% 体重下，与完整踝关节相比，ATFL 缺陷踝关节的上平移略有增加 0.2 ± 0.2 毫米 (p = 0.02)。 ATFL 缺陷会增加距骨的前平移、内旋和上平移。运动学的改变可能会导致慢性外侧踝关节不稳定性所观察到的退行性变化。这些发现可能有助于解释慢性 ATFL 功能不全患者内侧距骨上经常观察到的退行性变化，并为改善踝关节韧带重建以恢复正常关节运动提供基线。
Previous studies have suggested injury to the anterior talofibular ligament may be linked to altered kinematics and the development of osteoarthritis of the ankle joint. However, the effects of ATFL injury on the in vivo kinematics of the ankle joint are unclear. Based on the orientation of the ATFL fibers, we hypothesized that ATFL deficiency would lead to increased anterior translation and increased internal rotation of the talus relative to the tibia. Controlled laboratory study. The ankles of 9 patients with unilateral ATFL injuries were compared as they stepped onto a level surface. Kinematic measurements were made as a function of increasing load. Using magnetic resonance imaging and orthogonal fluoroscopy, the in vivo kinematics of the tibiotalar joint were measured in the ATFL deficient and intact ankles from the same individuals. A statistically significant increase in internal rotation, anterior translation, and superior translation of the talus was measured in ATFL deficient ankles as compared to intact, contralateral controls. For example, at 100% body weight, ATFL deficient ankles demonstrated a statistically significant increase in anterior translation of 0.9 ± 0.5mm (p = 0.008). At 100% body weight, the ATFL deficient ankle was internally rotated relative to the intact ankle by 5.7 ± 3.6° (p = 0.008). There was a slight increase of 0.2 ± 0.2mm in the superior translation of the ATFL deficient ankle compared to the intact ankle at 100% body weight (p = 0.02). ATFL deficiency increases anterior translation, internal rotation, and superior translation of the talus. Altered kinematics may contribute to the degenerative changes observed with chronic lateral ankle instability. These findings might help to explain the degenerative changes frequently observed on the medial talus in patients with chronic ATFL insufficiency and provide a baseline for improving ankle ligament reconstructions aimed at restoring normal joint motion.
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DOI：
10.1249/01.mss.0000222827.56982.40
发表时间：
2006-06-01
期刊：
MEDICINE AND SCIENCE IN SPORTS AND EXERCISE
影响因子：
--
作者：
Fujii, Tadashi;Kitaoka, Harold B.;An, Kai-Nan
通讯作者：
An, Kai-Nan
影响因子：
1.7
作者：
Hirose, K;Murakami, G;Yamashita, T
通讯作者：
Yamashita, T
DOI：
10.1097/01.blo.0000197079.26600.09
发表时间：
2006-01-01
影响因子：
4.2
作者：
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通讯作者：
Koo, Seungbum
DOI：
10.1097/01.blo.0000183737.43245.26
发表时间：
2006-01-01
影响因子：
4.2
作者：
Aydogan, Umur;Glisson, Richard R.;Nunley, James A.
通讯作者：
Nunley, James A.
影响因子：
18.4
作者：
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Hintermann, Beat