A prospective test of the dual-pathway model of bulimic pathology: mediating effects of dieting and negative affect.
A prospective test of the dual-pathway model of bulimic pathology: mediating effects of dieting and negative affect.
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DOI：
10.1037/0021-843x.110.1.124
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发表时间：
2001-02
影响因子：
4.6
通讯作者：
E. Stice
中科院分区：
文献类型：
--
作者：
E. Stice
由于对神经性贪食的综合病因学理论的纵向研究较少，本研究采用随机回归生长曲线模型和来自青春期女孩3波社区样本（N = 231）的数据，前瞻性地检验了双途径模型。最初的瘦压力和瘦理想内化预测了随后对身体不满的增长，最初的身体不满预测了节食和负面影响的增长，最初的节食和负面影响预测了暴食症症状的增长。大多数假设的中介作用都有前瞻性证据。结果与瘦的压力、瘦的理想内化、身体不满、节食和负面情绪是暴食症病理的危险因素的主张一致，并为双途径模型提供了支持。
Because there have been few longitudinal investigations of integrative etiological theories of bulimia nervosa, this study prospectively tested the dual-pathway model using random regression growth curve models and data from a 3-wave community sample of adolescent girls (N = 231). Initial pressure to be thin and thin-ideal internalization predicted subsequent growth in body dissatisfaction, initial body dissatisfaction predicted growth in dieting and negative affect, and initial dieting and negative affect predicted growth in bulimic symptoms. There was prospective evidence for most of the hypothesized mediational effects. Results are consistent with the assertion that pressure to be thin, thin-ideal internalization, body dissatisfaction, dieting, and negative affect are risk factors for bulimic pathology and provide support for the dual-pathway model.