Glioblastoma resistance to anti-VEGF therapy: has the challenge been MET?
Glioblastoma resistance to anti-VEGF therapy: has the challenge been MET?
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DOI：
10.1158/1078-0432.ccr-13-0051
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发表时间：
2013-04-01
期刊：
影响因子：
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通讯作者：
McCarty JH
中科院分区：
文献类型：
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作者：
McCarty JH
In glioblastoma cells the receptor tyrosine kinase c-Met is upregulated in response to Bevacizumab and plays an important role in promoting invasion and tumor recurrence. These data support novel links between VEGF-A and HGF and suggest that c-Met and its signaling effectors may be effective targets for anti-invasive therapies.
