MESONEPHROMA OVARII
MESONEPHROMA OVARII
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卵巢中肾病
DOI：
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发表时间：
1948
影响因子：
11.4
通讯作者：
H. Jacobs
中科院分区：
文献类型：
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作者：
H. Bettinger;H. Jacobs
---------- 1 ---------- 1——胰腺;因此，再次感染不可能是本质缺陷。因此，有必要提出第三个因素。3. 未知的因素。未知的因素可能是先天的或营养的。如果是先天性的，支气管的缺陷可能是Wolbach和Farber所描述的腺体结构全局性疾病的一部分。在营养方面，除了胰腺病变外，乳糜泻和纤维囊性疾病的病理差异（见表三）还在于后者的肝和肺紊乱。主要的代谢差异是蛋白质代谢，这在乳糜泻中是正常的，但在纤维囊性疾病中是紊乱的。这种蛋白质缺陷可能导致肝脏疾病；它不会引起肺部疾病吗？它可能与缺乏蛋白质抗体和缺乏对感染的抵抗力有关，有人认为，当纤维囊性疾病的营养缺陷得到补救时，可以推迟或可能避免支气管扩张的阶段。这个问题的答案只能在未来对这些孩子的研究中找到。下表（表III），在Andersen和Hodges（]2）的基础上稍作修改，概述了胰腺纤维囊性疾病和乳糜泻的区别。
----------1----------1--------pancreas; hence again infection cannot be the essential defect. It is thereforenecessaryto formulatea third factor. 3. Unknown factor. The unknown factor may be either congenital or nutritional. If congenital, the defect in the bronchi may be part of the generalizeddisorder of glandular structuresas outlined by Wolbach and Farber. With regard to nutrition, apart from the pancreatic lesion, the pathological differences between ceeliac disease and fibrocystic disease (see Table III) are in the liver and lung disturbances of the latter. The chief metabolic difference is in protein metabolism, which is normal in creliac diseasebut derangedin fibrocystic disease. This protein defect probably causesthe liver disorder; may it not also cause the lung disorder? It may be bound up with lack of protein antibodiesand lack of resistanceto infection, and it is being held that when the nutritional defect in fibrocystic disease is remedied, the stage of bronchiectasismay be postponedor possibly averted. The answer to this can be found only in the future study of these children. The following table (Table III), somewhatmodified from Andersen and Hodges, (]2) outlines the difference between fibrocystic diseaseof the pancreasand cceliac disease.