Pliometric contraction-induced injury of mouse skeletal muscle: Effect of initial length
Pliometric contraction-induced injury of mouse skeletal muscle: Effect of initial length
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DOI：
10.1152/jappl.1997.82.1.278
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发表时间：
1997-01-01
影响因子：
3.3
通讯作者：
Faulkner, JA
中科院分区：
文献类型：
--
作者：
Hunter, KD;Faulkner, JA
对于从最佳纤维长度（L（f））开始的单次测力（伸长）收缩，确定随后的力不足的最重要因素是拉伸期间的功输入。我们测试的假设，无论初始长度，力赤字主要是一个功能的工作输入。最大限度地激活小鼠趾长伸肌，并以2 L（f）/s的速度从三种初始纤维长度（L（f）的90、100或120%）之一延长到三种最终纤维长度（L（f）的150、160或170%）之一。在测力收缩之前和之后评估最大等长力产生。没有一个单一的机械因素，包括功输入（r（2）= 0.34），足以解释各组之间观察到的力缺陷的差异。因此，力的不足似乎是由机械事件的复杂相互作用引起的。对于按初始纤维长度分组的数据，平均功和平均力不足之间的相关性很高（r（2）= 0.97-0.99）。因此，最好的解释的基础上的初始纤维长度和拉伸过程中的工作输入组之间的力赤字的差异。
For single pliometric (lengthening) contractions initiated from optimal fiber length (L(f)), the most important factor determining the subsequent force deficit is the work input during the stretch. We tested the hypothesis that regardless of the initial length, the force deficit is primarily a function of the work input. Extensor digitorum longus muscles of mice were maximally activated in situ and lengthened at 2 L(f)/s from one of three initial fiber lengths (90, 100, or 120% of L(f)) to one of three final fiber lengths (150, 160, or 170% of L(f)). Maximal isometric force production was assessed before and after the pliometric contraction. No single mechanical factor, including the work input (r(2) = 0.34), was sufficient to explain the differences in force deficits observed among groups. Therefore, the force deficit appears to arise from a complex interaction of mechanical events. With the data grouped by initial fiber length, the correlation between the average work and the average force deficit was high (r(2) = 0.97-0.99). Consequently, differences in force deficits among groups were best explained on the basis of the initial fiber length and the work input during the stretch.