168
168
复制标题
168
DOI:
10.1097/01.ccm.0000550923.88428.dc
复制
发表时间:
2019
影响因子:
8.8
通讯作者:
K. Burchard
中科院分区:
文献类型:
--
作者:
P. Atchinson;M. Roginski;K. Burchard
Methods: A 36-year-old female presenting with Group A Streptococcus necrotizing fasciitis of the lower extremity developed hypotension and tamponade physiology three days after amputation of the lower extremity and massive fluid resuscitation with over 40 liters of crystalloid. She had initial hemodynamic improvement after amputation and antibiotics, however on day three developed rapidly progressive shock which was transiently fluid responsive. Initially, distributive shock due to sepsis was first on the differential, however tamponade was diagnosed with point of care echocardiography performed by the critical care physician. Pericardiocentesis was performed and fluid was consistent with a transudative effusion, without extension of infection or prior trauma to suggest alternate cause. Ultimately, tamponade was attributed to her large volume resuscitation in the setting of systemic inflammation.Results: This case report is relevant to the critical care community because it alerts us to the possibility of cardiac tamponade in the setting of massive fluid resuscitation, which is not well represented in current literature. Cardiac tamponade is an acutely life threatening condition, and traditionally described evidence of tamponade-Beck’s triad, pulsus paradoxus and Kussmaul’s sign-are unreliable indicators of tamponade physiology. Diagnosis relies on clinical suspicion and echocardiographic findings. Critical care clinicians with basic echocardiographic skills can readily identify the presence of a pericardial effusion at bedside, rapidly narrowing a broad differential and hastening definitive, life saving therapy.