RESPIRATORY AND CARDIOVASCULAR RESPONSE TO IMMERSION IN COLD AND WARM WATER
RESPIRATORY AND CARDIOVASCULAR RESPONSE TO IMMERSION IN COLD AND WARM WATER
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DOI：
10.1113/expphysiol.1961.sp001519
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发表时间：
1961-01-01
影响因子：
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通讯作者：
EVANS, M
中科院分区：
文献类型：
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作者：
KEATINGE, WR;EVANS, M
在浸泡在5摄氏度和15摄氏度的搅拌水中的最初几分钟里，12名裸体男子的肺通气量很高，他们的呼气末二氧化碳分压下降。二氧化碳分压随后恢复到或略高于其原始水平，但即使在持续20分钟的工作实验中也没有大大超过它。在5摄氏度或40分钟的水中保存。在15摄氏度的水中，虽然功减少或扭转了pCC2最初的下降，但这些结果并不能证明，在冷水中艰苦工作期间，至少在中等时间的浸泡中，pCC2会上升到危险水平。在37.8摄氏度的水中，S的心率稳步上升；在25摄氏度的水中，心率下降并保持在较低水平；在5摄氏度和15摄氏度的水中，心率先升后降。在15摄氏度的重复浸泡降低或消除了对浸泡的早期呼吸和心率反应以及代谢反应，但没有显著增加直肠温度的下降。衣服也降低了对沉浸的反射反应。在前2分钟内观察到大量的室性早搏。在15摄氏度的水中浸泡过的人，可能是由于静脉和动脉压升高、肾上腺素升高和过度换气导致的室颤，可能是一些冷水中猝死的原因。
During their first few minutes of immersion in stirred water at 5 and 15[degree] C the pulmonary ventilation of twelve unclothed men was high, and their end-tidal pCO2 fell. The pCO2 then returned to or a little above its original level but did not greatly exceed it even in working experiments lasting 20 min. in water at 5[degree] C or 40 min. in water at 15[degree] C. Although work reduced or reversed the initial fall in pCC2, these results therefore do not bear out predictions that the pCC2 would rise to dangerous levels during hard work in cold water, at least in immersions of moderate duration. In water at 37.8[degree] C the men''s heart rates rose steadily, in water at 25[degree] C they fell and remained low, and in water at 5 and 15[degree] C they rose and then fell. Repeated immersion at 15[degree] C reduced or abolished the early respiratory and heart rate responses to immersion and the metabolic response, but did not significantly increase the falls in rectal temperature. Clothing also reduced the reflex responses to immersion. A number of ventricular extrasystoles were observed during the first 2 min. of immersion in water at 15[degree] C, and it is suggested that ventricular fibrillation due to increased venous and arterial pressures, adrenaline, and hyperventilation, may be responsible for some cases of sudden death in cold water.