The role of caudate nucleus and corpus callosum atrophy in trauma-induced anterior horn dilation.
The role of caudate nucleus and corpus callosum atrophy in trauma-induced anterior horn dilation.
复制标题
尾状核和胼胝体萎缩在创伤性前角扩张中的作用。
作者：
C. Anderson;E. D. Bigler
对30例创伤性脑损伤（TBI）患者进行了损伤当天计算机断层扫描（CT）扫描和损伤后（至少6周）磁共振（MR）成像比较。尽可能匹配尾状核（CN）头部（灰质结构）和侧脑室前角（AH）的扫描。测量了CN和AH的表面积和胼胝体（CC）的宽度。结果显示CN表面积无显著变化，而AH显示显著增加。CC显示宽度显著减小。CN无明显变化，CC宽度明显减少，提示AH扩张可能是由于周围白色而不是灰质萎缩。
Day-of-injury computerized tomography (CT) scans were compared to post-injury (at least 6 weeks) magnetic resonance (MR) imaging of 30 traumatic brain-injured (TBI) patients. Scans were matched as closely as possible at the head of the caudate nucleus (CN), a grey matter structure, and the anterior horns (AH) of the lateral ventricle. The CN and AH surface areas and width of the corpus callosum (CC), a white-matter structure, were measured. Results demonstrated a non-significant change in CN surface area, while AH showed a significant increase. CC showed a significant decrease in width. Lack of significant change in the CN, combined with a significant decrease in CC width, suggests that AH dilation is probably due to surrounding white as opposed to grey-matter atrophy.
阿尔茨海默病患者和对照组的计算机断层扫描和磁共振成像扫描的比较分析。
影响因子：
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作者：
Sandor,T;Jolesz,F;Tieman,J;Kikinis,R;Jones,K;Albert,M
通讯作者：
Albert,M