ABSCISIC-ACID-DEFICIENT MUTANTS AT THE ABA GENE LOCUS OF ARABIDOPSIS-THALIANA ARE IMPAIRED IN THE EPOXIDATION OF ZEAXANTHIN
ABSCISIC-ACID-DEFICIENT MUTANTS AT THE ABA GENE LOCUS OF ARABIDOPSIS-THALIANA ARE IMPAIRED IN THE EPOXIDATION OF ZEAXANTHIN
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DOI：
10.1111/j.1365-3040.1991.tb01531.x
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发表时间：
1991-08-01
影响因子：
7.3
通讯作者：
TAYLOR, IB
中科院分区：
文献类型：
--
作者：
DUCKHAM, SC;LINFORTH, RST;TAYLOR, IB
测定了豌豆和拟南芥野生型和脱落酸（阿坝）缺陷突变体的叶黄素含量。豌豆的萎蔫突变体与非突变体对照没有区别。与此相反，植物纯合的突变等位基因在拟南芥的阿坝基因座是非常不同的野生型。在这些突变体中，玉米黄质积累到异常高的水平。主要的类胡萝卜素，紫黄质和9 '-顺式-新黄质几乎不存在的突变体色谱图。由此得出结论，阿坝遗传损伤损害了玉米黄质到紫黄质的环氧化，并且这导致不能积累阿坝。这为玉米黄质是阿坝的前体提供了明确的证据。
The xanthophyll content of wild type and abscisic acid (ABA) - deficient mutants of pea and Arabidopsis thaliana was determined. The wilty mutant of pea was indistinguishable from the non-mutant control. In contrast, plants homozygous for mutant alleles at the aba locus of Arabidopsis were very different from wild type. In these mutants, zeaxanthin accumulated to abnormally high levels. The major carotenoids, violaxanthin and 9'-cis-neoxanthin were virtually absent from the mutant chromatograms. It was concluded that the aba genetic lesion impairs the epoxidation of zeaxanthin to violaxanthin and that this results in an inability to accumulate ABA. This provides clear evidence that zeaxanthin is a precursor of ABA.