Microscale spatial distribution and health assessment of PM2.5-bound polycyclic aromatic hydrocarbons (PAHs) at nine communities in Xi'an, China
Microscale spatial distribution and health assessment of PM2.5-bound polycyclic aromatic hydrocarbons (PAHs) at nine communities in Xi'an, China
复制标题
中国西安九个社区PM2.5结合多环芳烃(PAHs)的微尺度空间分布及健康评估
DOI：
10.1016/j.envpol.2016.08.058
复制
发表时间：
2016
影响因子：
8.9
通讯作者：
Qian Zhang
中科院分区：
文献类型：
--
作者：
Hongmei Xu;Steven Sai Hang Ho;Meiling Gao;Junji Cao;Benjamin Guinot;Kin Fai Ho;Xin Long;Jingzhi Wang;Zhenxing Shen;Suixin Liu;Chunli Zheng;Qian Zhang
研究了2013年夏季Xi细颗粒物（PM2. 5）中多环芳烃（PAHs）的空间分布特征。在9个城市和郊区社区同时收集的24小时综合空气样品中，对16种优先考虑的多环芳烃进行了定量。多环芳烃的总质量浓度范围为32.4 - 104.7 ng m−3，平均值为57.1 ± 23.0 ng m−3。在郊区社区观察到的多环芳烃浓度（平均值：86.3 ng m−3）高于城市社区（平均值：48.8 ng m−3），这是由于在城市范围内更好地执行污染控制政策，同时郊区对机动车和大规模建筑物的管理混乱。克里格插值分析表明，工业化地区（Xi西部和西北部）的PAH水平较高。基于卫星的土地利用的视觉解释也适用于支持多环芳烃的空间分布之间的社区。在采样期间，9个群落的平均苯并[a]芘当量毒性（[BaP]eq）为6.9 ± 2.2 ng m− 3，显示出与多环芳烃水平基本相似的空间分布。根据PM2.5 [BaP]方程计算的过量吸入终生致癌风险表明，Xi市每百万社区居民中有8人因PM2.5结合的多环芳烃暴露而患癌症。多环芳烃在城市内的巨大空间变异性证实了多点采样进行暴露健康风险评估的重要性。
Spatial variability of polycyclic aromatic hydrocarbons (PAHs) associated with fine particulate matter (PM2.5) was investigated in Xi'an, China, in summer of 2013. Sixteen priority PAHs were quantified in 24-h integrated air samples collected simultaneously at nine urban and suburban communities. The total quantified PAHs mass concentrations ranged from 32.4 to 104.7 ng m−3, with an average value of 57.1 ± 23.0 ng m−3. PAHs were observed higher concentrations at suburban communities (average: 86.3 ng m−3) than at urban ones (average: 48.8 ng m−3) due to a better enforcement of the pollution control policies at the urban scale, and meanwhile the disorganized management of motor vehicles and massive building constructions in the suburbs. Elevated PAH levels were observed in the industrialized regions (west and northwest of Xi'an) from Kriging interpolation analysis. Satellite-based visual interpretations of land use were also applied for the supporting the spatial distribution of PAHs among the communities. The average benzo[a]pyrene-equivalent toxicity (Σ[BaP]eq) at the nine communities was 6.9 ± 2.2 ng m−3during the sampling period, showing a generally similar spatial distribution to PAHs levels. On average, the excess inhalation lifetime cancer risk derived from Σ[BaP]eqindicated that eight persons per million of community residents would develop cancer due to PM2.5-bound PAHs exposure in Xi'an. The great in-city spatial variability of PAHs confirmed the importance of multiple points sampling to conduct exposure health risk assessment.