Vestibular involvement in a passive transport and return task
Vestibular involvement in a passive transport and return task
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前庭参与被动运输和返回任务
DOI：
10.3758/bf03326763
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发表时间：
1983
期刊：
影响因子：
--
通讯作者：
L. Abraham
中科院分区：
文献类型：
--
作者：
Sue B. Miller;M. Potegal;L. Abraham
训练老鼠在被被动地沿着包含90度转弯的小路离开水嘴后回到水嘴上。通过改进的滴定程序，路径长度连续增加到132厘米。在摘除眼球后，该任务成功地重新学习。与小脑皮质损伤的影响相比，随后的前庭核损伤在这一任务中产生了严重的、持久的缺陷。相比之下，前庭病变在嗅觉定位任务中只产生轻微的一过性损伤。前庭最前部的病变也会影响空气恢复。
Rats were trained to return to a water spout after being passively transported away from it along paths containing a 90-deg turn. Path lengths were successively increased to as much as 132 cm by a modified titration procedure. The task was successfully relearned after enucleation. Subsequent vestibular nucleus damage produced a severe, lasting deficit in this task when compared with the effects of cerebellar cortex lesions. In contrast, the vestibular lesions produced only a mild, transient impairment in an olfactory localization task. The most anterior vestibular lesions also affected air righting.