Magnesium toxicity as a cause of hypotension and hypoventilation. Occurrence in patients with normal renal function.
Magnesium toxicity as a cause of hypotension and hypoventilation. Occurrence in patients with normal renal function.
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镁中毒是导致低血压和通气不足的原因。
DOI：
10.1001/archinte.1985.00360090068012
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发表时间：
1985
影响因子：
--
通讯作者：
John J. Marini
中科院分区：
文献类型：
--
作者：
C. Fassler;R. Rodriguez;David B. Badesch;W. Stone;John J. Marini
症状性高镁血症通常要求离子摄入量增加和肾功能异常；然而，我们治疗了两例肾功能正常的医源性高镁血症患者(10.4和13.2Meg/L)。其中一人接受了半酸性蛋白(Renacidin)的输尿管冲洗以溶解结石，另一人因摄入未知毒素而接受大剂量硫酸镁治疗。治疗包括呼吸机支持、静脉补钙和补液。不需要透析，恢复完全。
Symptomatic hypermagnesemia usually requires both increased intake of the ion and abnormal renal function; however, we treated two patients with iatrogenic hypermagnesemia (10.4 and 13.2 mEg/L) who had normal renal function. One received ureteral irrigation with hemiacidrin (Renacidin) to dissolve a stone, and the other was treated for ingestion of an unknown toxin with large doses of magnesium sulfate. Therapy included ventilatory support, intravenous calcium, and fluids. Dialysis was not required, and recovery was complete.