Protein palmitoylation and pathogenesis in apicomplexan parasites.

Protein palmitoylation and pathogenesis in apicomplexan parasites.
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DOI:
10.1155/2012/483969
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发表时间:
2012
影响因子:
--
通讯作者:
Caballero MC
Caballero MC
中科院分区:
其他
文献类型:
--
作者:
Corvi MM;Alonso AM;Caballero MC

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顶复门寄生虫包括多种原生动物寄生虫,包括刚地弓形虫、疟原虫、艾美耳球虫和隐孢子虫物种。作为细胞内的寄生虫,成功建立致病机制依赖于它们感染宿主细胞并在其中复制的能力。已知蛋白质棕榈酰化影响细胞生物学的许多方面。此外,棕榈酰化最近已被证明影响T。如复制、侵入和滑行。因此,本文着重于蛋白棕榈酰化在顶复门寄生虫发病机制中的重要性。
Apicomplexan parasites comprise a broad variety of protozoan parasites, including Toxoplasma gondii, Plasmodium, Eimeria, and Cryptosporidium species. Being intracellular parasites, the success in establishing pathogenesis relies in their ability to infect a host-cell and replicate within it. Protein palmitoylation is known to affect many aspects of cell biology. Furthermore, palmitoylation has recently been shown to affect important processes in T. gondii such as replication, invasion, and gliding. Thus, this paper focuses on the importance of protein palmitoylation in the pathogenesis of apicomplexan parasites.
弓形虫 IMC 蛋白的一个新家族在协调寄生虫分裂中表现出层次结构和功能。
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