Mechanisms of floral repression in Arabidopsis.
Mechanisms of floral repression in Arabidopsis.
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DOI：
10.1016/s1369-5266(02)00014-6
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发表时间：
2003-02
影响因子：
9.5
通讯作者：
Z. Sung;Lingjing Chen;Y. Moon;K. Lertpiriyapong
中科院分区：
文献类型：
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作者：
Z. Sung;Lingjing Chen;Y. Moon;K. Lertpiriyapong
在过去的两年中，一些早花基因已被证明编码假定的染色质相关蛋白在拟南芥。这些蛋白质可能作为表观遗传沉默子，抑制开花和花器官身份基因的促进，从而维持营养生长。随着植物的成熟，成花启动子的水平增加，尽管持续存在成花阻遏物。高水平的成花启动子在某种程度上能够克服成花抑制并激活花发育。进一步表征的突变体，无论是花的启动子或花的阻遏物中有障碍，这些突变体不仅显示缺陷的开花时间，但也有改变花序结构。这些结果表明，这些开花基因也调节其他方面的芽发育，并可能被用来研究芽生长模式的机制。
In the past two years, several early-flowering genes have been shown to encode putative chromatin-associated proteins in Arabidopsis. These proteins probably function as epigenetic silencers that repress the promotion of flowering and flower organ identity genes, and thereby maintain vegetative growth. As the plant matures, levels of the floral promoters increase despite the continued presence of floral repressors. High levels of the floral promoters are somehow able to overcome floral repression and to activate flower development. Further characterization of mutants that have impairments in either floral promoters or floral repressors revealed that these mutants not only display defects in flowering time but also have altered inflorescence architectures. These findings indicate that these flowering genes also regulate other aspects of shoot development and may be used to study the mechanism of shoot growth pattern.