HISTOPATHOLOGY OF NATURAL CARIES AROUND SILVER AMALGAM FILLINGS
HISTOPATHOLOGY OF NATURAL CARIES AROUND SILVER AMALGAM FILLINGS
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DOI：
10.1159/000260123
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发表时间：
1974-01-01
期刊：
影响因子：
4.2
通讯作者：
BIE, T
中科院分区：
文献类型：
--
作者：
HALS, E;ANDREASSEN, BH;BIE, T
本文研究了与银汞合金充填物有关的自然继发龋的组织病理学，并与以往文献中描述的实验性体外和体内继发龋的组织病理学进行了比较。该材料包括49个提取的人类恒牙，共55个银汞合金，主要是V类，填料。在宏观检查，约75%的牙齿显示初始继发龋，而25%的牙齿表面没有变化，相邻的填充物。将牙齿切成60-120 µm厚的纵向切片，通过偏振光和显微放射照相术进行检查。因为每个完整的切片都包括咬合/切牙和牙颈部腔壁与牙齿表面的相邻区域，所以它为组织学检查提供了两个“继发性龋齿风险”。然而，在处理各款的过程中，原来的风险数量有所减少。原则上，病变的组织病理学与实验性继发龋的组织病理学密切对应，但描述了某些差异。在釉质中，外部病变的频率为79.5%，在牙骨质中为90.9%。牙釉质和牙本质/牙骨质的腔壁病变的频率分别为69.5%和86.7%。与室壁病变相关的外部病变的发生率几乎是无室壁病变的外部病变的三倍。牙本质壁的不透射线性增加发生在27.8%的风险中，而牙釉质壁没有这种变化。在21个几乎没有或没有肉眼可见病变的风险组中，显微镜下的变化令人惊讶地频繁。
The histopathology of natural secondary caries associated with silver amalgam fillings was studied and compared with the histopathology of experimentalin vitroandin vivosecondary caries described in previous publications. The material consisted of 49 extracted human permanent teeth with a total of 55 silver amalgam, mostly class V, fillings. On macroscopical examination, about 75% of the teeth showed initial secondary caries, whilst 25% revealed no change of the tooth surface adjacent to the filling. The teeth were cut into longitudinal sections, 60–120 µm thick, which were examined by polarized light and microradiography. Because every complete section included an occlusal/incisal and a cervical cavity wall with an adjacent area of the tooth surface, it offered two ‘secondary caries risks’ for histological examination. However, the original number of risks was somewhat reduced during processing of sections. In principle, the histopathology of the lesions corresponded closely to that of the experimental secondary caries, but certain differences are described. In the enamel the frequency of outer lesions was 79.5%, in the cementum 90.9%. The frequency of cavity wall lesions in enamel and dentine/cementum was 69.5 and 86.7%, respectively. Outer lesions associated with a wall lesion were almost three times more frequent than outer lesions without a wall lesion. Increased radiopacity of the dentinal wall occurred in 27.8% of the risks, whilst no such change could be registered in the enamel walls. In the 21 risks of the group that had shown little or no macroscopical lesions, the microscopical changes were surprisingly frequent.