Carbon dioxide potentiates the mitogenic effects of nicotine and its carcinogenic derivative, NNK, in normal and neoplastic neuroendocrine lung cells via stimulation of autocrine and protein kinase C-dependent mitogenic pathways.

Carbon dioxide potentiates the mitogenic effects of nicotine and its carcinogenic derivative, NNK, in normal and neoplastic neuroendocrine lung cells via stimulation of autocrine and protein kinase C-dependent mitogenic pathways.
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二氧化碳通过刺激自分泌和蛋白激酶 C 依赖性有丝分裂途径,增强尼古丁及其致癌衍生物 NNK 在正常和肿瘤性神经内分泌肺细胞中的促有丝分裂作用。

DOI:
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发表时间:
1994
期刊:
影响因子:
3.4
通讯作者:
Schuller,HM
Schuller,HM
中科院分区:
医学3区
文献类型:
--
作者:
Schuller,HM

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