Dampening NF-κB signaling by "self-eating".
Dampening NF-κB signaling by "self-eating".
复制标题
通过“自噬”抑制 NF-κB 信号传导。
DOI：
10.1016/j.immuni.2012.06.008
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发表时间：
2012
期刊：
影响因子：
32.4
通讯作者：
Lin,Xin
中科院分区：
文献类型：
--
作者：
Blonska,Marzenna;Lin,Xin
NF-κB转录因子的激活对T细胞的存活、增殖和分化至关重要。在本期《免疫》杂志中，Paul等人（2012）证明自噬是tcr激活的NF-κB被翻转的途径。
Activation of NF-κB transcription factor is crucial for survival, proliferation, and differentiation of T cells. In this issue ofImmunity, Paul et al. (2012) demonstrate that autophagy is a pathway by which TCR-activated NF-κB is turned over.