Pathology of the posterior tibial tendon in posterior tibial tendon insufficiency
Pathology of the posterior tibial tendon in posterior tibial tendon insufficiency
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DOI：
10.1177/107110079801900803
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发表时间：
1998-08-01
影响因子：
2.7
通讯作者：
Manoli, A
中科院分区：
文献类型：
--
作者：
Mosier, SM;Lucas, DR;Manoli, A
对15例正常人和15例手术治疗的胫后肌腱进行了大体和组织学检查。所有手术标本均异常，内踝远端增大，外观呈暗白色。在组织学检查中，15例尸体肌腱中有12例显示正常肌腱结构，其特征为胶原束线性取向、正常成纤维细胞构成、低血管密度和插入软骨样化生。手术标本表现为退行性肌腱病，其特征为粘蛋白含量增加、成纤维细胞增生、软骨样化生和新生血管形成。这导致胶原束的线性取向明显中断。
Gross and histologic examinations of 15 normal cadaver and 15 surgical posterior tibial tendons from patients with posterior tibial tendon insufficiency were performed. All surgical specimens were abnormal with enlargement distal to the medial malleolus and a dull white appearance. At histologic examination, 12 of 15 cadaver tendons displayed normal tendon structure characterized by linear orientation of collagen bundles, normal fibroblast cellularity, low vascular density, and insertional chondroid metaplasia. The surgical specimens displayed a degenerative tendinosis characterized by increased mucin content, fibroblast hypercellularity, chondroid metaplasia, and neovascularization. This resulted in marked disruption of the linear orientation of the collagen bundles.