Deficiency of mouse FHR-1 homolog, FHR-E, accelerates sepsis and acute kidney injury through enhancing the LPS-induced alternative complement pathway
Deficiency of mouse FHR-1 homolog, FHR-E, accelerates sepsis and acute kidney injury through enhancing the LPS-induced alternative complement pathway
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DOI：
10.3389/fimmu.2020.01123
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发表时间：
2020
影响因子：
7.3
通讯作者：
李巍
中科院分区：
文献类型：
--
作者：
栗相如;郝振华;刘小荣;李巍
