Myofascial Trigger Points Then and Now: A Historical and Scientific Perspective.
Myofascial Trigger Points Then and Now: A Historical and Scientific Perspective.
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当时和现在的肌筋膜触发点：历史和科学的观点。
DOI：
10.1016/j.pmrj.2015.01.024
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发表时间：
2015-07
期刊：
影响因子：
--
通讯作者：
Gerber L
中科院分区：
文献类型：
--
作者：
Shah JP;Thaker N;Heimur J;Aredo JV;Sikdar S;Gerber L
本文的目的是从历史和科学的角度探讨肌筋膜触发点(MTrP)在肌筋膜疼痛综合征(MPS)中的作用。MTrP是骨骼肌紧绷带中的坚硬、离散、可触及的结节，可能自发疼痛(即活动)，或仅在受压时疼痛(即潜伏)。MPS是一个术语，用来描述一种疼痛状况，这种状况可以是急性的，或者更常见的是慢性的，涉及肌肉及其周围的结缔组织(如筋膜)。根据Travell和Simons的说法，MTrp是该综合征的核心--但它们是必要的吗？尽管在过去的两个世纪里，对肌肉疼痛和MTrP的临床研究激增，但科学文献似乎往往脱节和令人困惑。不幸的是，许多术语、理论、概念和诊断标准不一致、不完整或有争议。为了解决这些不足，研究人员最近应用临床、(骨骼肌和脑)成像和生化分析来系统和客观地研究MTrP及其在MPS中的作用。资料表明，MTrP周围的软组织环境、神经源性炎症、敏感化和边缘系统功能障碍都可能在MPS的启动、扩大和持续中发挥作用。作者将记录导致目前对MTrP病理生理学及其与MPS的关系的理解的进展，并回顾临床医生和研究人员的贡献，他们影响和扩大了我们当代的临床知识和实践水平。
The intent of this paper is to discuss the evolving role of the myofascial trigger point (MTrP) in myofascial pain syndrome (MPS) from both a historical and scientific perspective. MTrPs are hard, discrete, palpable nodules in a taut band of skeletal muscle that may be spontaneously painful (i.e. active), or painful only on compression (i.e. latent). MPS is a term used to describe a pain condition which can be acute or, more commonly, chronic and involves the muscle and its surrounding connective tissue (e.g. fascia). According to Travell and Simons, MTrPs are central to the syndrome—but are they necessary? Although the clinical study of muscle pain and MTrPs has proliferated over the past two centuries, the scientific literature often seems disjointed and confusing. Unfortunately, much of the terminology, theories, concepts, and diagnostic criteria are inconsistent, incomplete, or controversial. In order to address these deficiencies, investigators have recently applied clinical, imaging (of skeletal muscle and brain), and biochemical analyses to systematically and objectively study the MTrP and its role in MPS. Data suggest that the soft tissue milieu around the MTrP, neurogenic inflammation, sensitization, and limbic system dysfunction may all play a role in the initiation, amplification, and perpetuation of MPS. The authors will chronicle the advances that have led to the current understanding of MTrP pathophysiology and its relationship to MPS, and review the contributions of clinicians and researchers who have influenced and expanded our contemporary level of clinical knowledge and practice.