A mechanism of noncontinuous supraosseous tooth eruption.
A mechanism of noncontinuous supraosseous tooth eruption.
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DOI：
10.1067/mod.2001.116086
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发表时间：
2001-09
期刊：
影响因子：
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通讯作者：
T. Katona;Haihong Qian
中科院分区：
文献类型：
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作者：
T. Katona;Haihong Qian
已经提出了许多理论来解释牙齿萌出的各个方面，但对于控制这些方面的一些潜在机制还没有普遍的共识。一个重要的尚未解决的问题是取代牙齿的动力的来源。我们认为骨上爆发力是由功能性颌骨变形引起的牙槽变形产生的。先前的研究使用静力平衡的基本方程来证明斜牙周韧带（PDL）纤维的伴随拉伸会产生可能导致骨上牙出牙的力。为了进行更严格和更广泛的分析，我们采用了有限元法（FEM）。考虑了三种功能载荷，但有限元计算强烈表明，所有颌骨变形都有助于牙齿挤压。研究还表明，随着拉伸的增加，PDL纤维的破坏可能会发生，这可以提供一种稳定爆发位置的机制。
Numerous theories have been propounded to explain the various aspects of tooth eruption, but no general consensus exists about some of the underlying mechanisms that govern these aspects. An important unresolved issue is the source of the motive forces that displace teeth. We proposed that supraosseous eruptive forces are generated by tooth socket distortions caused by functional jaw deformations. Previous studies used basic equations of static equilibrium to demonstrate that the concomitant stretching of the oblique periodontal ligament (PDL) fibers give rise to forces that may cause supraosseous tooth eruption. For a more rigorous and expanded analysis, we applied the finite element method (FEM). Three functional loads were considered, but the FEM calculations strongly suggested that all jaw deformations contribute to tooth extrusion. It was also demonstrated that the PDL fiber disruptions that are likely to occur with increased stretching can provide a mechanism to stabilize the erupted position.