Caudal cingulate infarction manifesting astasia.

Caudal cingulate infarction manifesting astasia.
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DOI:
10.1159/000358047
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发表时间:
2014-01
影响因子:
0.7
通讯作者:
Kobayashi A
Kobayashi A
中科院分区:
其他
文献类型:
--
作者:
Satow T;Komuro T;Kobayashi A

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摘要脑中风后,由于丘脑腹外侧区的病变而引起的失语症是一种罕见的临床症状,通常被称为丘脑性失语症。我们报告一例尾扣带回梗塞表现为失语症。一名58岁的男性无法坐,站和走(astasia)。四肢无明显运动无力。磁振造影显示脑梗塞位于扣带回尾侧,位于前垂直连合线与后垂直连合线之间。他的症状持续了1年,程度较轻。尾侧扣带回的病变可表现为失动。责任病变位于VCA和VPC线之间的扣带回,这可能对应于人类的尾侧扣带回区。我们应该记住,失语症可能是中风的一个主要症状。
Astasia is a rare presenting symptom of stroke, usually known as ‘thalamic astasia’, induced by a lesion in the ventrolateral thalamus. We report a case of caudal cingulate infarction manifesting astasia. A 58-year-old male presented with inability to sit, stand and walk (astasia). No apparent motor weakness was noticed in the extremities. MRI revealed cerebral infarction in the caudal cingulate gyrus, which was located between the vertical commissure anterior (VCA) line and vertical commissure posterior (VPC) line. His symptoms persisted for 1 year to a lesser degree. Lesions in the caudal cingulate gyrus can present with astasia. The responsible lesion is located in the cingulate gyrus between the VCA and VPC line, which might correspond to the caudal cingulate zone in humans. We should keep in mind that astasia can be a presenting symptom of stroke.