The over‐expression of Pim‐2 promote the tumorigenesis of prostatic carcinoma through phosphorylating eIF4B
The over‐expression of Pim‐2 promote the tumorigenesis of prostatic carcinoma through phosphorylating eIF4B
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DOI：
10.1002/pros.22693
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发表时间：
2013-09
期刊：
影响因子：
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通讯作者：
Ke Ren;X. Gou;Ming-Chao Xiao;Ming Wang;Chao-dong Liu;Zhao-bing Tang;Weiyang He
中科院分区：
文献类型：
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作者：
Ke Ren;X. Gou;Ming-Chao Xiao;Ming Wang;Chao-dong Liu;Zhao-bing Tang;Weiyang He
细胞实验发现Pim-2可能参与前列腺癌（PCA）的发生。Pim-2在PCA细胞中的抗凋亡机制尚不清楚。
Cell experiments have found Pim‐2 may take part in the tumorigenesis of prostatic carcinoma (PCA). More direct evidences are needed, and the detailed anti‐apoptotic mechanism of Pim‐2 in PCA cells is still unknown.