Subunit-selective mutagenesis of Glu-89 residue in human immunodeficiency virus reverse transcriptase. Contribution of p66 and p51 subunits to nucleoside analog sensitivity, divalent cation preference, and steady state kinetic properties.
Subunit-selective mutagenesis of Glu-89 residue in human immunodeficiency virus reverse transcriptase. Contribution of p66 and p51 subunits to nucleoside analog sensitivity, divalent cation preference, and steady state kinetic properties.
复制标题
人类免疫缺陷病毒逆转录酶中 Glu-89 残基的亚基选择性诱变。
DOI：
--
复制
发表时间：
1994
期刊：
影响因子：
--
通讯作者：
Prasad,VR
中科院分区：
文献类型：
--
作者：
Kew,Y;Qingbin,S;Prasad,VR
