Visual evoked potentials change as heart rate and carotid pressure change.
Visual evoked potentials change as heart rate and carotid pressure change.
复制标题
视觉诱发电位随着心率和颈动脉压力的变化而变化。
作者：
Barbara B. Walker;Curt A. Sandman
心血管活动和大脑之间的关系进行了探讨，通过记录视觉诱发电位从枕部区域的头皮在收缩压和舒张压（实验I）和在快速和缓慢的心跳在收缩压和舒张压（实验II）。当心率和颈动脉压正常波动时，视觉诱发电位发生显著变化，且左右半球的变化有显著差异。在各种心脏事件中，从右半球记录的诱发电位差异显著，而从左半球记录的则没有。在这两个实验中，右半球的差异主要是由于PI分量，这是在舒张压比收缩压大。目前的研究结果是一致的配方从行为研究表明，压力感受器的活动可以影响感官的摄入，并表明，大脑半球的专业化可能发挥重要作用的心脏事件，大脑和行为之间的关系。颈动脉压力。诱发电位。心率压力感受器当Granit和Kaada（1952）发现中枢神经系统可以控制来自肌梭的传入纤维时，感觉冲动只是从受体传递到皮层的早期概念被动摇了。从那时起，许多研究已经确定，大脑可以通过抑制或促进从外周感觉器官到皮层水平的任何地方的感觉传递来调节自己的感觉输入。事实上，据估计，10%的传入纤维有助于离心控制机制（利文斯顿，1976），模糊了“感觉”和“运动”机制之间的经典区别。脑干网状结构在感觉过程的离心控制中起重要作用。网状结构的电刺激和损伤（利文斯顿，1959; Hernanwen-Peon，1961）都可以促进或阻断相互作用。
The relationship between cardiovascular activity and the brain was explored by recording visual evoked potentials from the occipital regions of the scalp during systolic and diastolic pressure (Experiment I) and during fast and slow heartbeats at systolic and diastolic pressure (Experiment II). Visual evoked potentials changed significantly as heart rate and carotid pressure fluctuated normally, and these changes were markedly different in the right and left cerebral hemispheres. Evoked potentials recorded from the right hemisphere during various cardiac events differed significantly, whereas those recorded from the left did not. In both experiments, differences in the right hemisphere were due primarily to the PI component, which was larger at diastolic than at systolic pressure. The present findings are consistent with formulations from behavioral studies suggesting that baroreceptor activity can influence sensory intake, and suggest that hemispheric specialization may play an important role in the relationship between cardiac events, the brain and behavior. DESCRIPTORS: Carotid pressure. Evoked potentials. Heart rate, Baroreceptors. The early conception that sensory impulses are simply relayed from receptor to cortex was shaken when Granit and Kaada (1952) discovered that the central nervous systetn could control afferent fibers from muscle spindles. Since then, numerous studies have established that the brain can modulate its own sensory input by inhibiting or facilitating sensory transmission anywhere from the peripheral sense organs to the cortical level. In fact, it has been estimated that 10% of all afferent fibers contribute to centrifugal control mechanisms (Livingston, 1976), blurring the classical distinction between "sensory" and "motor" mechanisms. The brainstem reticular formation plays an important role in the centrifugal control of sensory processes. Both electrical stimulation and lesions (Livingston, 1959; Hernandez-Peon, 1961) of the reticuiar formation can facilitate or block interac